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The potential of GHK as a therapeutic agent for fibrosis, including combination strategies with antioxidants or anti-inflammatory agents, is also explored

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4.3.4 Organelle dysfunction CS also impairs the function of critical organelles central to ferroptosis regulation: (1) Mitochondrial Damage: CS stimulates DRP1-dependent mitochondrial fission, resulting in fragmented, dysfunctional mitochondria that overproduce reactive oxygen species (ROS)